WAYS TO OPTIMIZE REHABILITATION ACTIVITIES IN HEMORRHAGIC AND ISCHEMIC STROKE
DOI:
https://doi.org/10.17605/OSF.IO/GTDXMKeywords:
cerebrovascular pathology, hyperlipidemia, physical inactivityAbstract
The development of acute cerebral ischemia triggers pathobiochemical cascade reactions, the outcome of which is cerebral infarction (MI), which is formed by two mechanisms: necrotic cell death and apoptosis - programmed cell death. These modern pathogenetic concepts have made it possible to propose a sequence of stages »Based on their causal relationships. Each stage of the cascade is a target for the therapeutic effect of drugs, primarily with neuroprotective effects.